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This inhibitory interaction may contribute to observed reductions in motor activity and sleep-associated effects: Enhancement of GABA-A receptor-mediated currents in specific brain regions Simultaneous NMDA receptor blockade in cortical areas Possible coordination of excitatory/inhibitory balance during sleep-wake transitions Relationship to GABA-A receptor sites remains an area of active investigation MAPK Cascade and GILZ Homology – Cellular Stress Response Research suggests DSIP interacts with components of the mitogen-activated protein kinase (MAPK) cascade through structural homology with glucocorticoid-induced leucine zipper (GILZ), a protein that modulates inflammatory and stress responses[3]