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In HFD models, AX attenuates hepatic triglyceride and cholesterol accumulation by downregulating key lipogenic factors such as sterol regulatory element-binding protein 1c (SREBP-1c), acetyl-CoA carboxylase (ACC), fatty acid synthase (FAS), and stearoyl-CoA desaturase-1 (SCD-1), while upregulating β-oxidation-related genes, including carnitine palmitoyltransferase 1 (CPT1) and peroxisome proliferator-activated receptor alpha (PPARα) [73]
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doi: 10.1021/acssuschemeng.2c04422
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