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Addiction There is an abundance of literature implicating glutamatergic abnormalities in addiction.47,48 More recently, data are emerging suggesting a role of oxidative stress in the pathophysiology of addiction to drugs of abuse.32,49–51 Research has explored the modulation of glutamatergic pathways by NAC in pre-clinical models.52,53 N -acetylcysteine has been shown to reverse the decline in cystine–glutamate exchange through the cystine–glutamate antiporter and thereby assist in the restoration of glutamatergic pathways in addiction.32,52 These properties have made it a potential prospect for the treatment of addiction