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Conclusions In summary, this study demonstrates the first evidence that exogenous GABA mitigates TS-like behavior in TS model rats by modulating harmful intestinal flora and striatal GABA-glutamate metabolism, central inflammatory state, and directly enhancing neuroprotective metabolites

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Of note, the particular BPC 157 counteracting potential toward hyperkalemia might first encourage the similar results obtained with the intragastric KCl application (27 mEq/kg)—(hyperkalemia 7 mmol/L) and BPC 157 therapy protocol (i.e., peaked T waves, fully counteracted by BPC 157 application, applied 30 min before or 10 min after KCl [67])